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The reviewed publications included mechanistic studies, clinical trials and systematic reviews involving human subjects that report relevant findings on direct epigenetic evidence (such as DNA methylation levels), biochemical markers or clinical outcomes associated with disease activity and therapeutic response

In various pathological conditions implicating oxidative stress, ER functions are disturbed resulting in ER stress and initiation and propagation of unfolded protein response (UPR), which results in transient suppression of protein translation to allow recovery but leads to cell death when stress cannot be resolved (Soliman et al., 2019)