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Trauma surgeons and reconstructive specialists typically frame compromised healing in terms of mechanical stability, infection control, and vascular supply, while ferroptosis researchers often approach bone from a cell biology perspective without explicit consideration of surgical workflows, staged reconstruction, or clinical phenotype heterogeneity ( in vitro osteoblast studies, with comparatively limited direct investigation of fracture healing, non-union, infected bone defects, or reconstruction-specific contexts such as the induced membrane technique (IMT) ( This review synthesizes current knowledge on ferroptosis in compromised bone healing, examining its biochemical basis, the ferroptosis-permissive post-traumatic microenvironment, and evidence across clinical phenotypes including osteoporotic fractures, induced membrane technique, infected bone defects, and non-union

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DOI Miller DM, DeSilva D, Pickart L, Aust SD

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